19 and frustrated...

[thestonedkoala Junior Member Find all posts View Profile Close thestonedkoala]

Hello, I just hopped on these forums because I'm very concerned and think it's time to start finding some help with my ED problems.

I'm 19, I lift weights, play hockey, do MMA, and am very fit, yet I've struggled to get erections for a few years now. I've hooked up with a lot of girls, more than I can remember, yet I've had sex once (which sucked). It's not because I don't know how to get to that point, it's because I get limp dick... Every time. And it's embarrassing...

I used to get random boners and rock hard erections when I first started masturbating, but it's been many years since then and now when I masturbate I can get only kind of hard. A possible concern I've had with this was that I would use a rough grip and rush through it. I just started PE and edging but when I edge and try to take it slow I can never get to the PONR.

I've been trying a couple of different things over the last year. I took supplements like citrulline and pycnogenol in hopes that it would help, but I didn't notice any improvement in my EQ. I thought that it could possibly be a psychological issue, and maybe it is, but I've tried to overcome it and haven't been able to.

I'm still taking supplements, going to the gym and doing everything possible. I started doing PE two weeks ago and started edging the other day. I don't watch porn anymore and vouch not to (but without it I can't really get stimulated). I feel like my problem is likely psychological and could possibly be helped by edging but, like I said, I struggle to edge cause I can't get to the PONR without fapping hard and fast. I'm looking for opinions on what some of you guys that have gone through this think. Any advice is welcomed, thanks!

Edit: I'm also circumsized and have read that can be a cause of ED over time. I'm considering restoring my foreskin as well and would like to know if you think that would help.

[tmacdaddy Banned Find all posts View Profile Close tmacdaddy]

ur circumcision didn't cause your ed but maybe the porn did...stop fapping...also go to your doctor and get prescribed viagra if its that bad

[popol5169 Senior Member Member of the Month Nov 2014 Find all posts View Profile Close popol5169]

Did you get hard with porn?

Did you use steroids?

[Jay1983 Senior MemberMember of the Month Apr 2014, Sept 2016 Find all posts View Profile Close Jay1983]

No porn or chicken choking for two weeks.

[Saskman Senior Member Find all posts View Profile Close Saskman]

You sound very similiar to me. My erectile dysfunction started at the age of 16 or so, until then, I could remember rock hard erections including rock hard nocturnal erections. Overnight they died. At the age of 30, I finally found out I have a hormonal imbalance and low testosterone, as well as what is known as a venous leak (though studies suggest this could be due to low testosterone, studies also show that venous leak in majority of patients studies disappears with administration of HRT in hypogondal younger patients).

When you lift weights do you find yourself making equal improvements to those around you? I have also been active, follow a healthy diet almost religiously, yet could never make gains. Within a month of taking clomidiphene, I am making gains and my muscles are bulging like they never did before. I also get sore after workouts, something that rarely occurred prior.

I strongly suggest you to not be the next me, get help now. Go see a urologist and make sure you anaylze your own bloodwork as often doctors simply follow references ranges to diagnose.

If you need anything else message me.

[thestonedkoala Junior Member Find all posts View Profile Close thestonedkoala]

You sound very similiar to me. My erectile dysfunction started at the age of 16 or so, until then, I could remember rock hard erections including rock hard nocturnal erections. Overnight they died. At the age of 30, I finally found out I have a hormonal imbalance and low testosterone, as well as what is known as a venous leak (though studies suggest this could be due to low testosterone).

When you lift weights do you find yourself making equal improvements to those around you? I have also been active, follow a healthy diet almost religiously, yet could never make gains. Within a month of taking clomidiphene, I am making gains and my muscles are bulging like they never did before. I also get sore after workouts, something that rarely occurred prior.

I strongly suggest you to not be the next me, get help now. Go see a urologist and make sure you anaylze your own bloodwork as often doctors simply follow references ranges to diagnose.

If you need anything else message me.

I've made some good gains, but not near as fast as some of my other friends. I eat very healthy and a lot, yet putting on weight has always come a bit difficult to me. I've hardly considered seeing a doctor, mostly because I can't afford the visit and would feel uncomfortable talking to my parents about this. Any suggestions to what I should do about that? Thanks for the advice.

Did you get hard with porn?

Did you use steroids?

I don't get super hard with porn but I'll admit that I get my best erections with it. And no, I've never used steroids.

[zezeze Senior Member Find all posts View Profile Close zezeze]

(though studies suggest this could be due to low testosterone, studies also show that venous leak in majority of patients studies disappears with administration of HRT in hypogondal younger patients).

I think you are misinterpreting the studies. I don't think there's even a single study that suggest low T being the cause of venous leak. Also low T is rarely the cause of ED in general and HRT only gives moderate results.

[corageon Banned Find all posts View Profile Close corageon]

I think you are misinterpreting the studies. I don't think there's even a single study that suggest low T being the cause of venous leak. Also low T is rarely the cause of ED in general and HRT only gives moderate results.

Low DHT is a huge risk factor for erectile dysfunction, and really the only androgen necessary with regard to the vascular part of erectile function .

Endocrinology. 1995 Apr;136(4):1495-501.
Dihydrotestosterone is the active androgen in the maintenance of nitric oxide-mediated penile erection in the rat.

Lugg JA1, Rajfer J, González-Cadavid NF.
Author information


Abstract

Androgens are essential for the expression of normal libido in the male, but their role in the maintenance of the erectile response in humans is controversial. It has been shown previously in the rat that castration induces 1) loss of penile reflexes; and 2) considerable reduction in the erectile response to electric field stimulation (EFS) of the cavernosal nerve. Both of these effects can be reversed by testosterone replacement. The current study was performed to determine whether these testosterone effects are mediated via its conversion to dihydrotestosterone (DHT), and to what extent the synthesis of the mediator of penile erection, nitric oxide, is affected by castration and androgen replacement. Five-month-old rats were either castrated or left intact. The orchiectomized rats were implanted with SILASTIC brand silicon tubing (Dow Corning) containing testosterone or DHT with or without daily injections of the 5 alpha-reductase inhibitor finasteride. After 7 days, rats were submitted to EFS and the intracavernosal pressure was recorded. Castration reduced the EFS-induced erectile response by 50% in comparison with intact rats and testosterone restored this decrease to normal. When finasteride was given to these testosterone-treated castrate rats, erectile response was not restored. DHT was as effective as testosterone in restoring response to EFS in castrates and this effect was not decreased by finasteride. Nitric oxide synthase activity in the penile cytosol was measured by the arginine-citrulline conversion and was found to correlate with the EFS determinations. These results show that DHT is the active androgen in the prevention of erectile failure seen in castrated rats, and suggest that this effect may be mediated, at least partially, by changes in nitric oxide synthase levels in the penis.


PMID: 7534702 [PubMed - indexed for MEDLINE]

[zezeze Senior Member Find all posts View Profile Close zezeze]

Low DHT is a huge risk factor for erectile dysfunction, and really the only androgen necessary with regard to the vascular part of erectile function .

But you just quote a study done with rats? Again is there a study that directly suggest low T being the cause of VL? Is it not true that statistically low T accounts to very few cases of ed, mainly only for those with low libido. And is it not true that most studies concludes that HRT to low T only gives moderate results regarding erectile function, mostly helps with the libido. I read one specialist saying that the HRT to low T does not directly improve erectile function in any way, but only increases the desire for sex.

[Saskman Senior Member Find all posts View Profile Close Saskman]

I think you are misinterpreting the studies. I don't think there's even a single study that suggest low T being the cause of venous leak. Also low T is rarely the cause of ED in general and HRT only gives moderate results.

Well aside from being a victim of hypogonadism (I am guessing secondary but I will find out in September when my other bloodtests are discussed with me), I have done a lot of Googling:

Dramatic improvement of penile venous leakage upon testosterone administration.

http://www.ncbi.nlm.nih.gov/pubmed/16420241

Testosterone improves erectile function in hypogonadal patients with venous leakage.

http://www.ncbi.nlm.nih.gov/pubmed/18599885

I agree with you, low T is a rare case of ED. But remember, my ED started at 16, which is rare itself. Out of that age group, having hormonal problems is also rare, but less rare then someone much older who has other risk factors. For ED in teenagers, and I always consider myself a teenager even though I'm 31 in regards to when my problem started, the most common causes other then psychological are: vascular issues and hormonol disorders.

Can low testosterone cause venous leak? Well, what is a venous leak exactly? Or is what appears to be a venous leak on a penile doppler ultrasound always a proper diagnosis? No. Even anxiety can appear on a doppler ultrasound as a venous leak. The doppler ultrasound should not be considered conclusive in diagnosing a venous leak, without bloodwork and a throughout medical background investigation, it doesnt hold that much clout in ensuring that the venous leak is truly the problem of ED. It is very good in assessing penile damage, like those Peyronie sufferers.

The false diagnosis of venous leak: prevalence and predictors.
The false diagnosis of venous leak: prevalence and predictors. - PubMed - NCBI

So Ive read in studies on the NIH website, that those who have been diagnosed with venous leak have seen there venous leak no longer recognizable on a doppler ultrasound. Basically, it disappeared. I also understand everything is a case by case basis, no two people will be the same, though the odds of being young and relatively healthy, having hypogondadsm and receiving testosterone therapy has apparently seen very fruitful results.

[Saskman Senior Member Find all posts View Profile Close Saskman]

I've made some good gains, but not near as fast as some of my other friends. I eat very healthy and a lot, yet putting on weight has always come a bit difficult to me. I've hardly considered seeing a doctor, mostly because I can't afford the visit and would feel uncomfortable talking to my parents about this. Any suggestions to what I should do about that? Thanks for the advice.

well i cant guide you to the US health system as I am a canadian, but i suggest that you do speak to your parents about this. i cant imagine drawing blood at a lab to be overly expensive. maybe you can find insurance, as urologists and or endocrinologists are likely expensive there.

anyways, i was never that skinny. but for the equal amount (or more) effort I put into the gym, I never achieved those huge results or forming. my brother, who was always skinny, started to work out and became a body builder. i have very good discipline, yet nothing, though i never thought much of it until this year when i found myself sometimes being able to lift less weight despite taking in protein and working out hard! then the doctor revealed my abnormal T levels.

after only just over a month of taking perscription clomidiphene (olympians use it to cheat), I am already starting to bulk and my strength has already increased, as well as my energy levels. depending on how it goes or doesnt go, i may start TRT this year. it really makes me feel like I have a new body. while i cant prove that half my life ago this problem started, as i never did T tests, i can only speculate.

if youre problem is not going away, i do suggest you speak to your parents and get a bloodtest done.

if you have this problem as i did, it wont resolve itself and there is nothing you can do that you read on the internet to make it any better without medication.

[mannakonda Senior Member Find all posts View Profile Close mannakonda]

Try 21 days witout porn

[Pegasus AdministratorPE Gym EditorPEGym Hero ☺ Admin of the Month Mar 2015 Find all posts View Profile Close Pegasus]

How long have you been without porn?
Edging can be done back from ponr.

[benschmidt Senior Member Member of the Month July 2015 Find all posts View Profile Close benschmidt]

I don't watch porn anymore and vouch not to (but without it I can't really get stimulated). I feel like my problem is likely psychological and could possibly be helped by edging but, like I said, I struggle to edge cause I can't get to the PONR without fapping hard and fast. I'm looking for opinions on what some of you guys that have gone through this think. Any advice is welcomed, thanks!

Stopping on the porn is good. If you have masturbated with a hard grip and very quickly, you may have conditioned yourself to these stimuli. Taking a break from masturbation for a couple weeks, and then beginning again with a light grip, slower and with some kind of lubricant. You need to retrain your brain.

[corageon Banned Find all posts View Profile Close corageon]

But you just quote a study done with rats? Again is there a study that directly suggest low T being the cause of VL? Is it not true that statistically low T accounts to very few cases of ed, mainly only for those with low libido. And is it not true that most studies concludes that HRT to low T only gives moderate results regarding erectile function, mostly helps with the libido. I read one specialist saying that the HRT to low T does not directly improve erectile function in any way, but only increases the desire for sex.

Oh zezeze, you talk so much and know so little.

Andrologia. 2006 Feb;38(1):34-7.
Dramatic improvement of penile venous leakage upon testosterone administration. A case report and review of literature.

Yassin AA1, Saad F.
Author information



Abstract

The main effect of testosterone was long-time assumed to be on sexual interest and, indirectly, on erectile function. Newer insights demonstrate that testosterone deficiency impairs the anatomical, ultrastructural, biological and physiological/functional substrate of penile erection, which can be, at least in part, restored by normalization of plasma testosterone levels. This is a report on a 56-year-old man suffering from diabetes mellitus type II and metabolic syndrome, who had complaints of a severe erectile dysfunction because of venous leakage, confirmed by pharmaco-cavernosography. He was also testosterone deficient (1.8 ng ml(-1)). Upon testosterone administration his erectile function improved dramatically. Repeated cavernosography no longer showed venous leakage.


PMID: 16420241 [PubMed - indexed for MEDLINE]


J Sex Med. 2006 Jul;3(4):727-35.
Testosterone undecanoate restores erectile function in a subset of patients with venous leakage: a series of case reports.

Yassin AA1, Saad F, Traish A.
Author information



Abstract

INTRODUCTION:

Androgens are critical for maintaining penile structure and function and androgen deficiency alters the function of the corporal veno-occlusive mechanism in animal models. However, there are limited research and data supporting this association in humans.
METHODS:

Case reports of hypogonadal men (N = 12) with low plasma testosterone and moderate to severe erectile dysfunction are presented. Comorbidities varied, including diabetes mellitus type I or II, metabolic syndrome with possible related hypertension, dyslipidemia, or obesity. Oral phosphodiesterase type 5 (PDE5) inhibitor therapy did not improve erectile function. Each patient underwent baseline dynamic infusion pharmacocavernosometry and cavernosography revealing various degrees of corporal veno-occlusive dysfunction. The patients underwent treatment with 1,000 mg injectable testosterone undecanoate (Nebido) on day 1, followed by another injection after 6 weeks and every 3 months thereafter. Dynamic infusion pharmacocavernosography was repeated in all 12 patients after 3 months of treatment.
RESULTS:

Five of the 12 patients reported significant improvement in erectile function within 12-20 weeks of androgen treatment and are currently under follow-up. Compared with baseline pharmacocavernosography, repeat radiological studies in patients who reported improvement in erectile function did not show veins draining the corporal bodies. The patients who responded to androgens also noted improvement in sexual desire domain (International Index of Erectile Function [IIEF] scores increased from 4 +/- 0.7 to 8 +/- 0.3) and erectile function domain (IIEF scores increased from 6 +/- 2 to 24 +/- 1).
CONCLUSION:

The observations made in these limited series of case reports suggest that testosterone improved erectile function in hypogonadal patients by restoring veno-occlusive function. Prospective, multi-institutional, double-blind placebo-controlled trials in hypogonadal patients are indicated.


PMID: 16839330 [PubMed - indexed for MEDLINE]

Diagnosis and treatment of venous leakage: a curable cause of impotence. - PubMed - NCBI
Br J Urol. 1988 Feb;61(2):151-5.

Diagnosis and treatment of venous leakage: a curable cause of impotence.

• 1Department of Surgery, Hammersmith Hospital, London.

Abstract

Sixteen of 149 patients complaining of impotence of a non-neurogenic or endocrine aetiology and with a penile brachial index of greater than 0.7 failed to achieve an erection following intracavernosal injection of 30 mg papaverine and 1 mg phentolamine. All 16 were shown to have a significant venous leak using digital subtraction cavernosography. Nine of the 13 so far operated upon have had a full return of potency. Repeat cavernosography in three failures showed persistent leakage into crural veins in two and non-ligated superficial veins in one. Impotent men with a normal penile brachial index who fail to respond to papaverine and phentolamine have a venous leak, a curable cause of impotence. Surgical access should allow ligation of all identified leaking veins.




Williams G1, Mulcahy MJ, Hartnell G, Kiely E.


Author information

A

PMID: 3280081 [PubMed - indexed for MEDLINE]


I guess it's another case of nitric oxide VS adrenaline as well.



Inhibition of sympathetic vasoconstriction is a major principle of vasodilation by nitric oxide in vivo.

• [*=left]J Zanzinger,
  [*=left]J Czachurski and
  [*=left]H Seller

+Author Affiliations

• I. Physiologisches Institut, Universität Heidelberg, Germany.

Abstract

The objective of this study was to determine whether vasodilator effects of nitric oxide (NO) can be explained by the inhibition of vasoconstriction caused by peripheral sympathetic nerve activity (SNA) in vivo. For this purpose, we studied the effects of systemic inhibition of NO synthesis during experimental variation of SNA in anesthetized cats. Intravenous infusion of NG-nitro-L-arginine methyl ester (L-NAME, 10 mg/kg) in baroreceptor-intact animals (n = 6) caused increases in mean arterial blood pressure (MAP) from 105.8 +/- 3.4 to 192.0 +/- 4.3 mm Hg that were associated with slight decreases in preganglionic SNA recorded from the white ramus of the third thoracic segment. Higher SNA appeared in completely baroreceptor-denervated cats (n = 10) than in the intact cats, but no changes in nerve activity occurred after the subsequent administration of L-NAME. In contrast, MAP increased from 123.3 +/- 4.0 to 245.8 +/- 5.1 mm Hg. In baroreceptor-denervated cats, reversible suppression of peripheral SNA produced by cooling of the ventral surface of the rostral ventrolateral medulla oblongata (RVLM) caused significant hypotension (61.1 +/- 2.6 mm Hg) and almost completely reversed the hypertension caused by L-NAME (76.0 +/- 3.7 mm Hg). Intravenous administration of the alpha 1-adrenergic receptor antagonist prazosin after L-NAME reduced MAP to a similar extent. In contrast, hypertension induced by angiotensin II could not be reversed by RVLM cooling. The pressor effects of intravenously administered noradrenaline during RVLM cooling were markedly potentiated by L-NAME and attenuated by the NO-donor compound S-nitroso-N-acetylpenicillamine (SNAP).(ABSTRACT TRUNCATED AT 250 WORDS)
Role of adrenal hormones (adrenaline and DOCA) in experimental hypertension