Chronic prostatitis

[lucyvucy Member Find all posts View Profile Close lucyvucy]

Hello,


I have seen a doctor today, and was diagnosed with Chronic Prostatitis.

The rectal exam he gave was very painful and he mentioned my prostate was enlarged and inflammed.

He did not know how to treat the disease and said there are many causes. Simply ordered anti-inflammatory suppository (indomethacin)...

...I do not know who can treat chronic prostatitis, or if the prostate is pressing on these nerves and vessels innervating the prostate area

I have turteling in a constant state almost, and cannot achieve erection. numb penis as well

[Tamora Retired Super Moderator Member of the Month Aug 2015 PEGym Hero Find all posts View Profile Close Tamora]

Isn't it caused by infection rather than being a disease?

Normally a course of antibiotics?

[jockinthebox Retired Moderator Member of the Month May 2015PEGym Hero Find all posts View Profile Close jockinthebox]

Get an appointment or referral to a Urologist. They know best how to treat prostatitis. Avoid alcohol, in the meantime, as that irritates the prostate further. Undercarriage issues are never fun. I've never had it, but have friends who have. You'll have to watch your diet carefully, in the future. Good luck, bro!

[lucyvucy Member Find all posts View Profile Close lucyvucy]

well my laundry list of symptoms are bad, but I guess my understanding is it may or may not be from infection. he said I had it, but didn't know the cause, or how to treat it.

is it treatable in both bacterial and non bacterial instances?

[corageon Banned Find all posts View Profile Close corageon]

well my laundry list of symptoms are bad, but I guess my understanding is it may or may not be from infection. he said I had it, but didn't know the cause, or how to treat it.

is it treatable in both bacterial and non bacterial instances?

Prostate issues of all sorts tend to come about when you don't have the proper ratio of testosterone:estrogen..estrogen flares up the prostate and too much adrenaline can create symptoms that mimic prostate enlargement.

If it is an infection - you can use a cocktail of herbs to rid it quickly and safely..you can use like Olive Leaf Extract + goldenseal extract...also add Vitamin C or eat tons of fruit.

Given the erectile issues are comorbid with prostate complaints - it is advisable to see a Urologist .
They can order more blood work and testing if they feel it is necessary.

Also drinking Pomegranate Juice may help a ton.
Eating cruciferous vegetables (broccoli, cauliflower, cabbage) is also helpful in these circumstances; plus they are good for your heart!

Since your prostate is both a filter and a vital part of the reproductive system - anytime something happens to it or there is enlargement or constriction (including in vas deferans etc) - other issues such as with arousal and orgasm can occur..so once the prostate is healthy your other issues may begin to subside as well.

[lucyvucy Member Find all posts View Profile Close lucyvucy]

wow okay thanks for the detail answer. I will try all that. I guess it can't hurt. I am suppose to see a urology who understands prostatitis. As of now, the other doctor recommended suppository anti-inflammatory.

thoughts on the suppository?

[lucyvucy Member Find all posts View Profile Close lucyvucy]

I have had painful ejaculations months ago when I was able to get an erection. also, painful sex at times. But last two months I can't get hard enough for penetration.

Had a hard time holding blood in (the valve to trap blood?)
can this be evaluated somehow?

[lucyvucy Member Find all posts View Profile Close lucyvucy]

this cocktail incredients can be found where??

[Tamora Retired Super Moderator Member of the Month Aug 2015 PEGym Hero Find all posts View Profile Close Tamora]

this cocktail incredients can be found where??

I would have thought a good chemist/health-shop and the supermarket. Just guessing!

[corageon Banned Find all posts View Profile Close corageon]

I would have thought a good chemist/health-shop and the supermarket. Just guessing!

Good instincts.

[invermol Member Find all posts View Profile Close invermol]

I agree to find a good urologist and have analysis done on you. Try yourself if there is a frequent reason for having it. In my case it was my brother contaminated urine due to his paraplegic condition and we all used the same toilet. Once you get rid of the cause, use different toilet in my case, then it is most certainly won´t come back. Kegels are useful to have a sane prostate. Good luck.

[lucyvucy Member Find all posts View Profile Close lucyvucy]

ok thank you.

other than antibiotics and anti-inflammatory, what about how should I be laying down at night, sitting etc?

[John Dough Senior Member Find all posts View Profile Close John Dough]

Prostate issues of all sorts tend to come about when you don't have the proper ratio of testosterone:estrogen..estrogen flares up the prostate.

High copper and low zinc levels in the body are said to cause estrogen dominance and prostate problems. Copper can be reduced by taking supplements of zinc and molybdenum, which are copper antagonists.

Copper Toxicity - Metabolic Healing

Among men, symptoms of copper toxicity, usually, include prostate enlargement, prostate infections and to some degree prostate cancer. Others include ED or erectile dysfunction , depression, anxiety

[Tamora Retired Super Moderator Member of the Month Aug 2015 PEGym Hero Find all posts View Profile Close Tamora]

Gosh...two pharmacists...wonderful!

[corageon Banned Find all posts View Profile Close corageon]

High copper and low zinc levels in the body are said to cause estrogen dominance and prostate problems. Copper can be reduced by taking supplements of zinc and molybdenum, which are copper antagonists.

Yes, that's true..but too much Zinc is just as bad...you can have nerve damage from Copper but also with Zinc; so stick to 15mg-25 mg a day at most...unless you are compensating with a little Copper as well - in an 8:2 ratio.

Usually micrograms of Copper even have profound effects on behavior...also high copper levels generally mold a more chaotic atmosphere..so people with elevated Copper are often impulsive and aggressive...sometimes even callous.. a lot of serial killers have shown elevated Copper and / or Lead , for example.

It hasn't been extensively tested though under the 'right' paradigms - but certainly Copper raises E2 levels which further contributes to hostility as estrogen is shown to be the driving force behind offensive male aggression...on the other hand..Androgens ; the most manly of hormones can actually have calming / anti-aggressive effects...

Physiol Behav. 1999 Jan 1-15;65(4-5):823-8.
Androgens and estrogens modulate 5-HT1A and 5-HT1B agonist effects on aggression.

Cologer-Clifford A1, Simon NG, Richter ML, Smoluk SA, Lu S.
Author information



Abstract

Intermale offensive aggressive behavior is facilitated by gonadal steroids and inhibited by serotonin (5-HT), presumably through its effects at 5-HT1A and 5-HT1B receptor sites. To examine the interaction between these neuroendocrine and neurochemical regulatory systems, CF-1 male mice were gonadectomized and implanted with silastic capsules containing either diethylstilbestrol (DES, a synthetic estrogen), the nonaromatizable androgens methyltrienolone (R1881) or dihydrotestosterone (DHT), or testosterone (T). Two weeks later, they were given 8-hydroxy-2-(di-n-propylamino)tetralin (8-OH-DPAT, a 5-HT1A agonist; 0.1 or 1.0 mg/kg), CGS12066B (a 5-HT1B agonist; 4.0 or 8.0 mg/kg), 0.1 or 1.0 mg/kg 8-OH-DPAT + 4.0 mg/kg CGS12066B, or vehicle, and tested for aggression. In the presence of DES, the higher 8-OH-DPAT dose given in combination with CGS attenuated aggression in comparison to vehicle controls. When given nonaromatizable androgen (R1881 or DHT), all drug treatments except 0.1 mg/kg 8-OH-DPAT significantly reduced offensive attack behavior. In the presence of T, which provides estrogenic and androgenic stimulation, aggression scores were significantly reduced when males were given the high dose of 8-OH-DPAT or CGS12066B, as well as in the 1.0 mg/kg 8-OH-DPAT + CGS12066B condition. Assessments of changes in motor behavior showed significant impairment when 8.0 mg/kg CGS12066B was administered across all hormonal conditions, indicating that reductions in offensive aggression in these treatment groups were nonspecific. The results demonstrate differential effects of the steroidal environment on the ability of 5-HT1A and 5-HT1B agonists to modulate aggression, with estrogens producing a more restrictive environment than androgens for serotonergic inhibition of male-typical aggressive behavior.


PMID: 10073487 [PubMed - indexed for MEDLINE]

What that means is androgens, the ones incapable of converting into estrogen; DHT and 3-alpha-diol - these can quell aggression in the presence of serotonin...on the other hand..high levels of serotonin are associated with more 'instrumental aggression' e.g obsessive-compulsive disorders, religious crimes and other 'divine' forms of aggression..so high serotonin can certainly provide an aggressive environment..just a different type of aggression than what would present with low serotonin.

http://psycnet.apa.org/journals/pne/7/4/601.html
Aggressive behavior has received considerable research attention for more than five decades. Although extensively studied, the mechanisms involved in both functional and pathological aggression are still far from elucidated. The regulation of aggression by a wide spectrum of neurotransmitters is well known. Serotonin has shown both inhibitory and stimulating effects on aggressive behavior, depending on the brain region measured and specific receptors where it acts. Dopamine and the mesocorticolimbic system associated with reward seeking behavior are also associated with aggression. Dopamine can sometimes enhance aggression and sometimes reduce the impulsivity that might lead to abnormal aggression. γ-Aminobutyric acid (GABA) is the main inhibitory neurotransmitter, and its relationship with aggressive behavior is extremely complex and highly associated with serotonin. This review focuses on summarizing the roles played by these three neurotransmitters (serotonin, dopamine, and GABA) in aggressive behavior and analyzing aggressive behavior from both neuropsychology and interdisciplinary perspectives.


Neurosci Biobehav Rev. 1998;23(2):325-36.
Testosterone and its metabolites modulate 5HT1A and 5HT1B agonist effects on intermale aggression.

Simon NG1, Cologer-Clifford A, Lu SF, McKenna SE, Hu S.
Author information


Abstract

Our understanding of the neurochemical and neuroendocrine systems' regulating the display of offensive intermale aggression has progressed substantially over the past twenty years. Pharmacological studies have shown that serotonin, via its action at 5HT1A and/or 5HT1B receptor sites, modulates the display of intermale aggressive behavior and that its effects serve to decrease behavioral expression. Neuroendocrine investigations, in turn, have demonstrated that male-typical aggression is testosterone-dependent and studies of genetic effects, metabolic function and steroid receptor binding have shown that facilitation of behavioral displays can occur via independent androgen-sensitive or estrogen-sensitive pathways. Remarkably, there have been virtually no studies that examined the interrelationship between these facilitative and inhibitory systems. As an initial step toward characterizing the interaction between the systems, studies were conducted that assessed hormonal modulation of serotonin function at 5HT1A and 5HT1B receptor sites. They demonstrated: (1) that the androgenic and estrogenic metabolites of testosterone differentially modulate the ability of systemically administered 8-OH-DPAT (a 5HT1A agonist) and CGS12066B (a 5HT1B agonist) to decrease offensive aggression; and (2) when microinjected into the lateral septum (LS) or medial preoptic area (MPO), the aggression-attenuating effects of 1A and 1B agonists differ regionally and vary with the steroidal milieu. In general, the results suggest that estrogens establish a restrictive environment for attenuation of T-dependent aggression by 8-OH-DPAT and CGS 12066B, while androgens either do not inhibit, or perhaps even facilitate, the ability of 5HT1A and 5HT1B agonists to reduce aggression. Potential mechanisms involved in the production of these steroidal effects are discussed and emerging issues that may impact on efforts to develop an integrative neurobiological model of offensive, intermale aggression are considered.


PMID: 9884126 [PubMed - indexed for MEDLINE]



A shorter version - even though I hate generalizations...DHT and 3-alpha-diol can reduce aggression when serotonin is sufficient..they may even improve serotonin's ability to reduce aggression..whereas estrogen blocks serotonin's effects on some receptors/brain regions..leading to more capacity for aggression and impulsivity..however, this does not apply in the same manner to say; schizophrenia or delusion based erratic reactions..as hormones are not as implicated and elevated serotonin can precipitate psychotic, religio-aggressive and Vigilante type thinking.